Ncaa defensive statistics: College Football – Defensive Rankings & Statistics

2022 College Football Complete Stats – Defense

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Defense

Player Player on team	GP Games played	INT Interceptions	SCK Sacks
M. Fuqua S BUFF Marcus Fuqua S BUFF	13	7	0
D. Patterson CB MTSU Decorian Patterson CB MTSU	13	7	0
M. Abraham DB MRSHL Micah Abraham DB MRSHL	13	6	0. 5
Y. Banks S SALA Yam Banks S SALA	13	6	1
S. Brown DB ILL Sydney Brown DB ILL	12	6	1
E. Forbes CB MISSST Emmanuel Forbes CB MISSST	12	6	0
K. Kinchens S MIAMI Kamren Kinchens S MIAMI	12	6	0
B. Morrison CB ND Benjamin Morrison CB ND	11	6	0
C. Phillips III CB UTAH Clark Phillips III CB UTAH	12	6	1
J. Taylor II S OKLAST Jason Taylor II S OKLAST	13	6	0
C. Bullock DB USC Calen Bullock DB USC	14	5	0
C. Chattman S TXSA Clifford Chattman S TXSA	14	5	0
B. Clark S TCU Bud Clark S TCU	12	5	0
C. DeJean DB IOWA Cooper DeJean DB IOWA	13	5	0
C. Lockridge DB FRESNO Cam Lockridge DB FRESNO	13	5	—
Q. Mitchell CB TOLEDO Quinyon Mitchell CB TOLEDO	12	5	0
R. Rahimi S LIB Robert Rahimi S LIB	13	5	—
B. Sanders DB NEVADA Bentlee Sanders DB NEVADA	12	5	0
K. Smith DB ILL Kendall Smith DB ILL	12	5	0
J. Stanley S USM Jay Stanley S USM	12	5	—
J. Torchio S WISC John Torchio S WISC	13	5	1
J. Brents DB IOWA Julius Brents DB IOWA	13	4	0
C. Coldon CB WYO C.J. Coldon CB WYO	10	4	0
J. Ford LB TEXAS Jaylan Ford LB TEXAS	12	4	2
C. Gonzalez DB OREG Christian Gonzalez DB OREG	12	4	0
Q. Johnson DB MEMP Quindell Johnson DB MEMP	12	4	0
I. Larsen S UTAHST Ike Larsen S UTAHST	10	4	2
K. Lovely CB WMICH Keni-H Lovely CB WMICH	12	4	—
D. McGlothern DB ARK Dwight McGlothern DB ARK	13	4	0
R. Moore DB MICH Rod Moore DB MICH	13	4	0. 5
T. Nubin DB MINN Tyler Nubin DB MINN	11	4	0
W. Roberts DB LATECH Willie Roberts DB LATECH	12	4	0
K. Rodgers CB TXSTSM Kordell Rodgers CB TXSTSM	9	4	0
J. Skinner S BOISE JL Skinner S BOISE	12	4	0
J. Thomas S MTSU Jakobe Thomas S MTSU	9	4	0
B. Trahan S UL Bralen Trahan S UL	13	4	0
Q. White CB GAST Quavian White CB GAST	10	4	1
A. White CB NCST Aydan White CB NCST	13	4	1
T. Wortham DB UCONN Tre Wortham DB UCONN	13	4	0
D. Carnell DB MIZZOU Daylan Carnell DB MIZZOU	13	3	0
R. Cooper Jr. DB OREGST Ryan Cooper Jr. DB OREGST	13	3	1
C. Edmonds DB ARIZST Chris Edmonds DB ARIZST	12	3	0
W. Johnson DB MICH Will Johnson DB MICH	11	3	0
S. Lockett III DB WASHST Sam Lockett III DB WASHST	11	3	0
D. Mask DB UCF DeJordan Mask DB UCF	12	3	0
C. Mason S KSTATE Cincere Mason S KSTATE	10	3	0
M. Shorts DB USM Malik Shorts DB USM	13	3	—
R. Texada CB NTEXAS Ridge Texada CB NTEXAS	13	3	0
D. Toombs II S FAU Dwight Toombs II S FAU	12	3	0
J. Walker LB JMAD Jailin Walker LB JMAD	10	3	—

College Football | PFF

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Age-related macular degeneration

Age-related macular degeneration (AMD) is one of the most common eye diseases and is the leading cause of vision loss in people over 40 years of age. According to the World Health Organization, 161 million people in the world suffer from eye diseases, including 25-30 million people affected by AMD. In Russia, the incidence of age-related macular degeneration is more than 15 people per 1000 population, but to date there is a large number (up to 51.6%) of Russians who have never heard of AMD (Bausch & Lomb study on age-related macular degeneration 2007).

Patients diagnosed with AMD do not see objects that they are directly looking at, which makes it impossible to watch TV, drive a car, recognize familiar faces, straight lines become “wavy”, sometimes some letters become invisible when reading, color vision is reduced – all this disrupts the normal course of life for such people. Usually, AMD is discovered by accident and therefore too late, because the brain compensates for the initial loss of vision. Pain, as a sign that something is wrong, is absent in this disease.

Age-related macular degeneration is a progressive eye disease that leads to a decrease in visual acuity due to damage to the central part of the retina – the macula (millions of cones and rods are located tightly adjacent to each other). Macular degeneration usually affects both eyes, although it can be asymmetrical, meaning one eye is more affected than the other.

There are two forms of AMD: “dry” (or atrophic) degeneration, which occurs when the pigment density of the macula decreases and can turn into “wet” (or exudative). Although the “wet” form makes up only about 10-20% of all cases of AMD, it is often the cause of blindness. With this form of the disease, the ingrowth of newly formed vessels under the retina begins from the side of the choroid. These vessels form the subretinal neovascular membrane. Fluid leakage from the neovascular membrane leads to local detachment of the retinal layers and the inevitable death of photoreceptors – rods and cones. Protein exudate, hemorrhages aggravate the situation, leading to the formation of a rough scar in the macula and irreversible deterioration of vision.

Risk factors:

The older a person gets, the more vulnerable the protective system of the eyes becomes and requires additional help. Along with the age factor , which is a “natural” cause of AMD, there is also a risk group for the development of this disease.

Smoking : Smokers are twice as likely to develop AMD than non-smokers. The reason for this is the metabolic changes that affect the development of AMD. Smoking leads to the formation of free radicals. Smokers require higher doses of antioxidants that neutralize free radicals. Quit smoking or cut down on cigarettes as soon as possible!

Unbalanced diet : lack or deficiency in the diet of essential vitamins and trace elements, as well as protective carotenoids (lutein and zeaxanthin), weakens the body’s antioxidant system.

Obesity : Scientists have discovered that the deposition of two important substances, lutein and zeaxanthin, is carried out in adipose tissue and in the macula. Both carotenoids (plant pigments) are found in high concentrations in the adipose tissue of overweight people, while their content in the tissues of the eye was low. This problem cannot be solved by increasing the intake of lutein and zeaxanthin. Only weight loss can set the stage for successful supplementation.

Solar radiation and, above all, the blue part of the solar spectrum is the most dangerous for the retina and can lead to partial loss of vision. The function of natural “sunglasses” in our body is performed by the carotenoids found in the retina of the eye – lutein and zeaxanthin (these substances can only come with food, the human body is not able to produce them). They “filter” blue light and act as antioxidants. But with age, the amount of carotenoids, which are part of the antioxidant defense system, decreases.

AMD may also result from prolonged exposure to light on the retina. In this case – as with smoking – there is an additional and excessive formation of free radicals, which, in turn, cause an increase in the load on the protective system, so ophthalmologists recommend using sunglasses to protect the eyes.

Members of the ethnic group: people with fair skin and blue irises are more susceptible to AMD.

Gender : women are more susceptible to AMD than men.

Heredity : people with cases of AMD among close relatives.

Genetic factor : in some cases, a combination of genes is the cause of AMD.

If one eye is affected by AMD, there is a high chance that the disease will develop in the other eye as well.

The pathological process in age-related macular degeneration flows individually, however, with the development of the subretinal neovascular membrane, the time factor becomes of key importance. With early diagnosis of this condition and timely treatment, it is possible to avoid the loss of visual functions, achieve long-term remission (temporary attenuation of the process) or its reverse development.

In the prevention of the development of AMD, a balanced diet is of great importance, including fruits and green vegetables (cabbage, pepper, spinach, broccoli, green peas, corn), containing large amounts of antioxidant vitamins A and E, carotenoids and zinc, and the use of lutein-containing supplements leading to to an increase in the concentration of lutein in the blood serum and retina.

Treatment:

Treatment of AMD includes: monitoring, taking antioxidant vitamin and mineral supplements, intravitreal injections (into the vitreous body) of drugs (Lucentis, Eylea, Vizchiu) that inhibit (depress, suppress) the production of endothelial vascular growth factor (VEGF). It has been proven that when they are introduced into the vitreous body, the growth of blood vessels stops, exudation decreases, which contributes to the preservation of the nerve cells of the macula. Lucentis is injected into the vitreous body in a strictly defined dose. The operation is usually performed 3 times with an interval of 1 month. The condition of the macula is monitored using a high-resolution optical coherence tomography (OCT) scanner. Lucentis was the first drug, the treatment of which leads not only to a slowdown in the progressive decline in central vision, but also to a partial restoration of visual acuity (increased visual acuity in 25-40% of patients and its stabilization in 9 patients). 5%).

Another treatment for AMD is photodynamic therapy. It is based on the intravenous administration of a drug called verteporfin (Vizudin), which, being deposited in the walls of retinal vessels, selectively increases their sensitivity to laser radiation of a certain wavelength. Thus, a targeted effect on pathological newly formed vessels is achieved and the degree of damage to healthy surrounding tissues is reduced. In carefully conducted comparative studies, scientists have convincingly shown that in certain forms of wet AMD, photodynamic therapy can stop the progression of the disease and improve vision.

A few tips for patients:

If you have been diagnosed with age-related macular degeneration, this is not a reason to go into the disease. You can, albeit not much, but improve your condition in some simple ways:

• Regularly check your vision with the Amsler grating – this will allow you to detect changes in the retina in time that require treatment.
• Take a multivitamin with trace elements and lutein – this has been proven to slow down the development of the disease.
• Include leafy green vegetables in your diet: spinach, kale, lettuce.
• When in the sun, always wear UV protection goggles.
• Quit smoking. Smoking disrupts the functioning of blood vessels, including retinal vessels.
• Do not forget about physical exercises – this leads to a general improvement of the body and increases the efficiency of the vascular system.

Allergy to the sun – photodermatitis (photodermatosis) and its prevention – St. Petersburg GBUZ Dermatovenerologic Dispensary No. 11

The sun’s rays not only warm us with their warmth in spring and summer and delight us in winter, but also help to strengthen the immune system. But, unfortunately, in some people, exposure to the open sun can cause an allergic reaction.